Retraction

FOXO1 is crucial in glioblastoma cell tumorigenesis and regulates the expression of SIRT1 to suppress senescence in the brain

Paper Information

Record ID:
16668
Publication Date:
November 23, 2017
Retraction Date:
December 27, 2017 (7.9 years years ago)
Article Type:
Publisher:
Spandidos
Open Access:
Yes
PubMed ID:
Retraction PubMed ID:
Not indexed in PubMed

Retraction Details

Retraction Reason:

False/Forged Authorship

Nature of Retraction:

Retraction

Retraction Notice:
10.3892/mmr.2017.8356

Citations (8)

8
Total Citations
8
Post-Retraction
(100.0%)
0
Pre-Retraction
0
Same Day
Post-Retraction Citation Analysis
0 Within 30 days
3 Within 1 year
3 After 2+ years
2251 Days since retraction (latest)
Paper citing FOXO1 is crucial in glioblastoma cell tumorigenesi...
Unknown Authors
Unknown Journal
Published: Feb 2024
2251 days after retraction
Paper citing FOXO1 is crucial in glioblastoma cell tumorigenesi...
Unknown Authors
Unknown Journal
Published: May 2021
1221 days after retraction
Paper citing FOXO1 is crucial in glioblastoma cell tumorigenesi...
Unknown Authors
Unknown Journal
Published: Mar 2021
1160 days after retraction
Paper citing FOXO1 is crucial in glioblastoma cell tumorigenesi...
Unknown Authors
Unknown Journal
Published: May 2019
505 days after retraction
SIRT5 downregulation is associated with poor prognosis in glioblastoma
Xi Chen, Zhijie Xu, Shuangshuang Zeng et al. (11 authors)
Cancer Biomarkers
Published: Mar 2019
29 citations
447 days after retraction
Paper citing FOXO1 is crucial in glioblastoma cell tumorigenesi...
Unknown Authors
Unknown Journal
Published: Sep 2018
261 days after retraction
Paper citing FOXO1 is crucial in glioblastoma cell tumorigenesi...
Unknown Authors
Unknown Journal
Published: Mar 2018
80 days after retraction
Paper citing FOXO1 is crucial in glioblastoma cell tumorigenesi...
Unknown Authors
Unknown Journal
Published: Feb 2018
49 days after retraction
Quick Stats
Total Citations: 8
Years Since Retraction: 7.9 years
Open Access: Yes
Last Checked: Never
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