Retraction

Mechanisms of M2 Macrophage-Derived Exosomal Long Non-coding RNA PVT1 in Regulating Th17 Cell Response in Experimental Autoimmune Encephalomyelitis

Paper Information

Record ID:

33034

Author(s):

Lei Wu
Jinjin Xia
Donghui Li
Ying Kang
Wei Fang
Peng Huang

Journal:

Frontiers in Immunology ( Frontiers )

Publication Date:

September 04, 2020

Retraction Date:

September 24, 2021 (4.2 years years ago)

Subjects:

Broad Categories:
Neuroscience Biology Genetics

Specific Fields:
Biology - Cellular Biology - Molecular Genetics Neuroscience

Institutions:

Country:

🇨🇳 China

Article Type:

Research Article

Publisher:

Frontiers

Open Access:

Yes

DOI:

10.3389/fimmu.2020.01934

PubMed ID:

33013847

Retraction PubMed ID:

34630443

Retraction Details

Retraction Reasons:

Nature of Retraction:

Retraction

Retraction Notice:

10.3389/fimmu.2021.773900

Additional Notes:

see also: https://pubpeer.com/publications/49E21CF1C2C5BDFD3303A5B2D59CF0;

Citations (32)

32

Total Citations

22

Post-Retraction
(68.8%)

10

Pre-Retraction

0

Same Day

Post-Retraction Citation Analysis

1 Within 30 days

15 Within 1 year

5 After 2+ years

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Antinociceptive effects of macrophage-derived extracellular vesicles by carrying microRNA-216a.

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Quick Stats

Total Citations: 32

Years Since Retraction: 4.2 years

Open Access: Yes

Last Checked: Jul 24, 2025

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Mechanisms of M2 Macrophage-Derived Exosomal Long Non-coding RNA PVT1 in Regulating Th17 Cell Response in Experimental Autoimmune Encephalomyelitis

Paper Information

Institutions:

Country:

Retraction Details

Retraction Reasons:

Nature of Retraction:

Retraction Notice:

Additional Notes:

Citations (32)

32

22

10

0

Post-Retraction Citation Analysis

Integrated analysis of differentially expressed genes and a ceRNA network to identify hub lncRNAs and potential drugs for multiple sclerosis.

Antinociceptive effects of macrophage-derived extracellular vesicles by carrying microRNA-216a.

Exosomes containing miR-122-5p secreted by LPS-induced neutrophils regulate the apoptosis and permeability of brain microvascular endothelial cells by targeting OCLN.

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